A myasthenia gravis patient develops muscle paralysis because

1
the nerve terminal at the neuromuscular junction fails to release acetylcholine.
2
although enough acetylcholine is released at the neuromuscular junction, it is destroyed by acetylcholinesterase.
3
the patient develops immunity against his own acetylcholine receptor
4
the patient develops antibody against his own acetylcholine. 

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